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GSH participates in thiol-disulfide exchange through a reversible reaction catalyzed by thiol-transferases as shown below, where Protein-SSG is a mixed disulfide and Protein-SH is reduced protein: The thiol-disulfide equilibrium is determined by the redox state of the cell, which is based on GSH and GSSG concentrations

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Figure 2 2.1 Reproductive system inflammation in PCOS 2.1.1 Ovary SLCI contributes to defects in oocyte quality, leading to ovulatory infertility, and accelerating the decline in ovarian reserve ( in vitro fertilization and embryo transfer (IVF-ET) treatment, reflecting the local function and pathological state within the PCOS ovaries ( Pro-inflammatory TNF-, primarily secreted by macrophages, was significantly increased in the ovaries and FF of PCOS patients ( In vitro experiments demonstrated that TNF- can directly induce apoptosis of primary rat GCs after 24h of incubation in serum-free medium 24 ( in vivo treatment with TNF inhibits steroidogenesis of the thecal cells and GCs, leading to regression of the corpus luteal ( Another pro-inflammatory cytokine IL-6, which is produced by mononuclear (MNC) cells and adipose tissue cells

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